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Short saphenous territory - Anatomy of recurrences following saphenopopliteal junction ligation - Ectatic gastrocnemius veins
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Figure 101 : Recurrence involving
a perforating vein after saphenopopliteal junction ligation.
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The particular anatomical and hemodynamic characteristics
of the veins of the popliteal fossa are responsible for a number
of therapeutic difficulties.
Firstly, the anatomical difficulties of this region are related
to the confluence of popliteal and saphenous collecting vessels
and muscle draining veins.
The hemodynamic difficulties are due to the fact that the popliteal
fossa is the site of very intense pressure variations. The intramuscular
network constitutes a very large blood reservoir which is suddenly,
mobilized during muscle contraction. The pressure within the
popliteal network increases considerably in the case of impairment
of venous drainage, (postural compression, stretching, and breath-holding
effort).
These specific conditions account for the high recurrence rate
following saphenopopliteal junction ligation despite a correct
surgical procedure, flush with the popliteal vein.
Figure 101 shows a recurrence involving a perforating vein of
the popliteal fossa appearing 18 months after saphenopopliteal
junction ligation and short saphenous stripping. This type of
recurrence is observed all the more frequently when the gastrocnemius
veins are ectatic with a large intramuscular blood volume.
In terms of pathogenesis. prior to saphenopopliteal junction
ligation, reflux by the short saphenous vein constitutes a sort
of safety valve to control hypertension in the popliteal network
(Figure 102A).
After saphenopopliteal junction ligation flush with the popliteal
vein and short saphenous stripping (Figure 102B), hypertension
of the popliteal vein is all the more marked when the gastrocnemius
veins are dilated or when there is an obstruction to blood drainage
and a new safety valve will he found in the ectatic development
of a perforating vein of the popliteal fossa (Figure 102 C).
The rapid development of these recurrences clearly illustrates
that the source of venous hypertension and blood reflux is situated
in the popliteal region and not in gastrocnemius perforating
veins (Figure 102D).
Figure 102A |
Figure 102B |
Figure 102C |

Figure 102D |
 Further reading
Blanchemaison Ph. La physiopathologie veineuse. Phlébologie, 1995; 48:
87-8.
Davy A., Ouvry P. Recurrence of varicose veins. Phlebology, 1986; 1: 15-21.
Hobbs J. The treatment of venous disorders. MTP, Lancaster, 1977: 159-201.
Perrin M. L'insuffisance veineuse chronique des membres inférieurs. MEDSI,
Paris, 1990.
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